top of page
Infected patient in quarantine lying in

Protect the most vulnerable patients from blood flow impairment

A major reason patients are repositioned every two hours is to prevent blood flow impairment in the sacral region

Screen Shot 2021-01-14 at 8.54.11 AM.png

Blood flow impairment occurs because the blood vessels become compressed, restricting circulation

Research has demonstrated

a variety of harmful physiological effects resulting from compromised sacral region blood flow

Inflammatory mediator release

Increased pain and discomfort

Pressure injury formation

Traditional pressure redistribution beds, overlays, and cushions were not designed to prevent sacral-region vascular compression

2021_0226_TurnCare_Competitor Render_BED
2021_0226_TurnCare_Competitor Render_SEA

These surfaces were designed to spread pressure across pre-set locations without understanding the characteristics of the patients' sacral anatomy, the location of the bony prominences, or the physiology of human vasculature

Introducing Vasotactic™ technology

We at TurnCare developed a patented Vasotactic™ technology, which provides precise, anatomy-specific therapy.


Vasotactic™ support therapy prevents vessels in the sacral region from becoming compressed, thereby avoiding blood flow impairment.

2020_0119 Turncare Patient Rendering 120

A Different Approach


Support surface that knows bony prominence location




Non-repeating therapy algorithm that gives sacral vessels time to spring back to shape



Adapts uniquely to each patient and underlying surface in real time


Bharucha JB, Seaman L, Powers M, Kelly E, Seaman R, Forcier L, ... & Wang L. A prospective randomized clinical trial of a novel, noninvasive perfusion enhancement system for the prevention of hospital-acquired sacral pressure injuries. Journal of Wound Ostomy & Continence Nursing. 2018;45(4);310-318.


Doll DN, Barr TL, Simpkins JW. Cytokines: Their role in stroke and potential use as biomarkers and therapeutic targets. Aging and Disease. 2014;5(5);294-306.

Eltzschig HK, Eckle T. Ischemia and reperfusion–from mechanism to translation. Nature medicine. 2011;17:1391–1401.

Kalogeris T, Baines CP, Krenz, M, Korthuis, RJ. Cell biology of ischemia/reperfusion injury. Int Rev Cell Mol Biol. 2012;298;229-317. 

Needham DM. Mobilizing patients in the intensive care unit: improving neuromuscular weakness and physical function. JAMA. 2008;300:1685–1690.

Parker A, Sricharoenchai T, Needham DM. Early rehabilitation in the intensive care unit: preventing physical and mental health impairments. Curr Phys Med Rehabil Rep. 2013;1(4):307–314.

Payen D, Lukaszewicz AC, Legrand M, Gayat E, Faiyre V, et al. A multicentre study of acute kidney injury in severe sepsis and septic shock: Association with inflammatory phenotype and HLA genotype. PLos ONE. 2012;7(6);e35838.

Pierce SM, Skalak TC, Rodeheaver GT. Ischemia-reperfusion injury in chronic pressure ulcer formation: a skin model in the rat. Wound Repair and Regeneration. 2000;8(1);68-76.

Truong A, Fan E, Brower R, Needham D. Bench-to-bedside review: mobilizing patients in the intensive care unit—from pathophysiology to clinical trials. Crit Care. 2009;13:216.

Zhang JM, An J. Cytokines, inflammation, and pain. International anesthesiology clinics. 2007;45(2);27-37.

p: (203) 437-6768


a: 2100 Geng Rd.

    Suite 210 - #119

    Palo Alto, CA 94303

  • Facebook - White Circle
  • White Twitter Icon
bottom of page